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OUTER HOUSE, COURT OF SESSION
[2024] CSOH 56
A39/13, A63/13, A64/13, A65/13, A101/13, A102/13,
A103/13, A108/13, A115/13, A120/13, A122/13
OPINION OF LORD CLARK
In the cause
SIMON PELOSI AND OTHERS
Pursuers
against
LANARKSHIRE HOUSING ASSOCIATION LTD
Defender
Pursuers: Sutherland; Allan McDougall
Defender: Johnston KC, McKinlay; Brodies LLP
5 June 2024
Introduction
[1]
Thirty-six actions were raised against Lanarkshire Housing Association Ltd, by
individuals who claim to have suffered personal injury caused by contaminants present in
the land at a housing development. Each pursuer is, or was, a tenant in the development
and the defender is the landlord. The housing, which is in Motherwell, is known as "the
Watling Street development". This opinion deals with eleven of the actions, heard together,
and the outcome will be binding in respect of the remaining cases. The issues to be dealt
2
with are: (i) whether the defender has breached its statutory duties; and (ii) if so, whether
that has caused loss to the pursuers.
[2]
These cases first called more than ten years ago, in early 2013. They were then sisted
by judges for several years and, after a decision at an opposed motion hearing, a reclaiming
motion was dealt with. Following the decision of the Inner House, when the cases called
before me, parties were encouraged to have discussions about identifying the lead case or
cases. It took some time for that to be sorted out, but in due course it was agreed that
thirteen cases would be the lead cases, although two of these cases were later removed,
leaving the current eleven cases.
[3]
Thereafter, several case management hearings took place. Despite efforts to have the
actions determined reasonably soon, that turned out not to be possible. Lengthy periods of
adjustment were needed, along with extensive periods for the lodging of full medical
records of the numerous pursuers, a number of expert reports and supplementary reports.
That affected the fixing of the proof date. By way of example, three expert reports were only
able to be lodged in December 2023. Motions were made to discharge the proof but the
grounds were not persuasive and, having regard also to the need for a reasonably efficient
disposal of the cases, the motions were refused.
[4]
In due course the proof took place, over a period of just less than three weeks. Many
productions were lodged and evidence was led from the thirteen pursuers and six experts.
As a consequence, this opinion is more lengthy than normal, particularly because after
summarising the factual and expert evidence, and parties' submissions, many contentious
points have to be addressed, and that is done in my assessment of the expert evidence.
3
Glossary
[5]
The expert reports, evidence and submissions all make reference, using acronyms or
abbreviations, to types of solvent, chemicals and contaminants and to expressions used in
guidance documents. While the meaning of each acronym or abbreviation is set out in the
narrative of the evidence that follows, some of them are then referred to more than once, and
it may help to have the meanings of those terms readily to hand in this glossary.
TCE
Trichloroethane
PCE
Tetrachloroethylene
VOCs
Volatile organic compounds
SVOCs
Semi-volatile organic compounds
PAHs
Polycyclic aromatic hydrocarbons
PCBs
Polychlorinated biphenyls
COPC
Constituents of potential concern
MTBE
Methyl-tertiary butyl ether
UCL95
Upper confidence limit of the mean
GACs
Generic assessment criteria
SGVs
Soil guideline values
DEFRA
Department of Environment, Food and
Rural Affairs
4
Background
The site
[6]
The Watling Street development was built on a former industrial site in Motherwell.
It extended to approximately 10.6 hectares, divided into four parcels of land: Plot A, Plot B1,
Plot B2, and Plot C. The properties owned by the defender and tenanted by the pursuers are
built wholly within Plot A of the site.
[7]
The industrial site was developed from farmland at the beginning of the 20th century,
becoming the location of the Motherwell Iron and Steel Works. The firm operated there
until around 1940. The site was transferred to Metropolitan-Vickers in 1947. In the 1950s
the site was redeveloped as an engineering works, manufacturing control mechanisms for
the heating and ventilation industry. Metropolitan-Vickers was rebranded as AEI in 1960.
By 1969 the site was occupied by Satchwell Sunvic (another subsidiary of AEI, which made
heating controls). By the late 1970s work had ceased on the site and the buildings were, at
least in part, demolished. The site was earmarked for redevelopment by the Scottish
Development Agency, with planning permission granted by Motherwell District Council in
May 1988.
[8]
The defender is the owner and landlord of ninety-four properties on the site. The
total number of residents at the site was not the subject of evidence in this case, but it is
estimated that there may be around two hundred people living there. The thirteen pursuers
who gave evidence lived in ten different properties on the site.
Investigations carried out
[9]
Investigations that were carried out in the past are referred to and considered by
expert witnesses for each side and so are now briefly summarised. In the early 1990s, the
5
council instructed Scott Wilson Kirkpatrick (SWK) to carry out contamination studies, in
order to assess the cost of making the site ready for sale for residential use. Various
investigations were conducted or supervised by SWK and remediation works were done.
Put broadly, while various metals, chemicals, solvents and compounds were found, SWK
concluded that the degree of risk of harm from chemical contamination had been reduced to
an acceptably low-level consistent with the residential use of the site. Citylink
Developments Ltd carried out the development of Plot A, with construction commencing in
the late 1990s. In July 2000, the contractors and developers arranged for further testing of
certain back gardens, resulting in metals and solvents being found, causing the ground in
those particular back gardens to be removed and replaced with soil brought in. The houses
had, by then, been constructed.
[10]
Residents moved into the properties from 1999. In due course, they became
concerned about health problems and around 2009-10 many were made aware of the
previous industrial use of the site and the presence of potential contaminants. A resident
raised these concerns with North Lanarkshire Council. The Council instructed WSP UK Ltd
(WSP) to carry out investigations. WSP carried out various investigations between 2010
and 2012, including an environmental assessment, and further investigations described as
Stage 1, Stage 2 and Stage 3. These included groundwater sampling and taking soil samples
from 64 gardens, nominally three from each garden. WSP's assessment results, applying
generic assessment criteria (GACs), concluded that two gardens (2 Tiber Avenue and
12 Romulus Court) had a "hotspot" of polycyclic aromatic hydrocarbon (PAH)
concentrations which were above the residential screening criteria. One garden (18 Forum
Place) had two hotspots of PAH concentrations above the criteria. Another garden (6 Forum
6
Place) had two hotspots of lead and nickel concentrations above the criteria. These hotspots
were generally recorded at depths of 20 to 40 centimetres under the surface.
[11]
Other properties were also found by WSP to have shown the presence of
contaminants but these were, following analysis, found to fall below the level stated by
GACs for residential use. A recommendation was made that consideration be given to the
excavation and off-site disposal of the hotspot areas which had been identified, followed by
replacement with inert subsoil and topsoil suitable for residential use.
[12]
Solicitors for the pursuers instructed Monridge Environmental LLC, a company
owned and managed by Mr Brien, one of the expert witnesses for the pursuers. Monridge
carried out indoor air sampling at Cornelia Street, Empire Way, Forum Place, and Tiber
Avenue and gave their results in a report in August 2011. This was reviewed by WSP.
Monridge also did a report following investigations in relation to Empire Way, Forum Place,
and Tiber Avenue in June 2012 and then a Site Investigation and Vapour Intrusion
Assessment at 31 Empire Way in March 2012. Further investigations resulted in a report by
Monridge in April 2013. Twenty five properties were investigated by Monridge in their
indoor air quality investigation. IKM Consulting were also instructed by the pursuers'
solicitors and did a human health risk assessment at 31 Empire Way, reported on 18 May
2012, and a further investigation reported on 17 May 2013. WSP did a further investigation
at 31 Empire Way in an Indoor Air and Sub-Slab Soil Vapour Assessment, reported on
5 November 2013.
Metals, chemicals, solvents and compounds on the site
[13]
The made ground on which the properties were built contained brick, ash, clinker,
various metals and their derivatives, and a variety of volatile organic compounds (VOCs),
7
semi-volatile organic compounds (SVOCs), polycyclic aromatic hydrocarbons (PAHs), and
possibly polychlorinated biphenyls (PCBs). Made ground is an area where material is
known to have been placed on the pre-existing land surface. The remediation of the site for
residential development did not remove all made ground. In the ground and groundwater
at Plot A of the site, where the pursuers' houses were constructed, there are compounds
present, which include carbon tetrachloride, TCE, PCE, benzene, benzo(a)pyrene, acetone,
propanol, 2-propanol, ethanol, cis-1,2 dichloroethene and toluene.
[14]
As a consequence, the chemical and physical properties of the contaminants in the
ground (except for metals) have the potential to evaporate. The extent to which an
individual contaminant will evaporate depends on temperature and the properties of the
contaminant. Chemical and physical properties of VOCs and SVOCs mean that they can
break down over time to other forms of compound. The distribution of the compounds
mentioned above within the site, including contamination by VOCs, SVOCs, PCBs and
metals, is not uniform. Potential pathways for transmission of the compounds to the
pursuers include inhalation of vapour fumes emitted by the compounds in the soil and
groundwater, and from direct or dermal contact with the compounds in the soil.
Statutory provisions
[15]
Section 113 of Housing (Scotland) Act 1987, and Schedule 10, paragraph 2, provide
that:
"In any contract to which this paragraph applies there shall, notwithstanding any
stipulation to the contrary, be implied a condition that the house is at the
commencement of the tenancy, and an undertaking that the house will be kept by the
landlord during the tenancy, in all respects reasonably fit for human habitation ..."
Section 27 of the Housing (Scotland) Act 2001, and Schedule 4, paragraph 1, provide that:
8
"The landlord in a Scottish secure tenancy must--
(a)
ensure that the house is, at the commencement of the tenancy, wind and
watertight and in all other respects reasonably fit for human habitation, and
(b)
keep the house in such condition throughout the tenancy."
The 2001 Act applied to tenancies commencing on or after 30 September 2002. Five of the
pursuers were tenants prior to then, with the remaining eight moving in thereafter.
However, the requirements for habitation in the provisions are almost identical.
Factual Evidence
The pursuers
[16]
Evidence was led from the thirteen pursuers, in this order: Barbara Strachura,
Elizabeth Butler, Marie Wilson, Donald Glen, Mary Adams, Joseph Tougher, Fiona Tougher,
Kathleen MacDonald, Nicoletta Kovacs, Simon Pelosi, Theresa Ward, Lee Brannan, and
Laura McCluskey. They each gave witness statements and their medical records were
lodged. Eight of them had also completed an indoor air assessment survey in 2011. There
were many common points in the evidence they gave and they also had to disclose
information of a personal nature, so it is not necessary, or appropriate, to set out each
pursuer's evidence in detail.
[17]
Some of the pursuers lived on the ground floor of their building, others on the first
floor and some in mid-terrace or end-terrace properties. The properties are located within
the development, in Forum Place, Tiber Avenue, Cornelia Street and Empire Way. One
pursuer moved into her property in 1999, four in 2001, one in 2006, four in 2008, two in 2009
and one in 2011. Six of them no longer live there.
9
[18]
In examination-in-chief, counsel for the pursuers, Mr Sutherland, followed a similar
approach with each pursuer. They each adopted their witness statements, their responses to
a medical questionnaire and other documents which they had completed. The pursuers
explained what they would smell within the properties and the gardens, and, in some detail,
their own symptoms and ailments. Mr Sutherland then took each witness to what the expert
instructed for the defender (Professor Eddleston) said about them. This included having
certain of the symptoms before they moved into the house, there being symptoms now
stated but which they had not mentioned to their GP or were not contained in the GP
records, and symptoms they still experienced after having left the property. The pursuers
were asked to comment on these points and did so. There was no cross-examination of any
of the pursuers.
[19]
Their references to experiencing a smell often described it as strong, unpleasant,
sweet and gassy or oily. For some it occurred throughout the year, both inside and outside
the house. Others spoke to it being stronger in specific parts of the house and at different
times. Some said that when flooring was being replaced the smell was even stronger. A
number of them had some of the symptoms they referred to before moving in, but described
them as generally not as bad as what was experienced when living there. Some explained
why they had not gone to their GP or had not stated certain things to the GP, largely
because, as they put it, they just had to get on with it and could use over-the-counter
medication. Occasionally a pursuer said they did tell their GP about symptoms, even
though that was not recorded. Several pursuers explained symptoms stopping or at least
improving when they moved out or were away from the property, sometimes weeks or
months later. They also commented on poor soil conditions and difficulties in gardening.
10
[20]
The key symptoms referred to were, as listed and described in the pursuers' written
submissions: (a) recurring or continuing headaches; (b) lethargy, exhaustion, tiredness and,
for some, depression; (c) inflammation of the eyes, ears or throat; (d) joint pain or stiffness;
(e) confusion or fatigue; (f) pins and needles; (g) stomach problems; (h) rashes and
dermatitis and (i) problems with the heart, lung or kidney. No-one had all of these ailments,
but each had several of them. The evidence about the last one, (i), was fairly limited. A
number of the pursuers mentioned other health issues, some of which are perhaps related to
certain of the symptoms listed. This included eczema, UTI, IBS, blocked nose, nose bleeds,
sinus problems, dizziness, bowel problems, hay fever, asthma, itchiness, vomiting and
diarrhoea. One witness said that anti-depressants were taken, all to do with the property.
Another one mentioned mould in the house. Family members or visitors were referred to by
certain witnesses as suffering some of the symptoms. The pursuers each explained when
they were first told that the land was contaminated, generally in 2010 or 2011.
Agreed evidence of other witnesses
[21]
The evidence of two other witnesses was agreed by the parties. Andrew McFarlane
of DM Hall was instructed for the pursuers and gave a report in 2012 in connection with a
particular property (31 Empire Way) in respect of routes for potential vapour transfer, along
with two short supplementary reports, one of which commented on other premises.
Gavin Young, who at the time was the Technical Services Manager for the defender, gave a
witness statement explaining, among other things, the checks and repairs carried out at
properties on the site. The evidence of these witnesses was not relied upon by either side as
being of any real significance to the key issues in the case.
11
Expert evidence
[22]
It is convenient at this stage to briefly summarise the main points made by the
experts. Further details of their opinions and reasoning, relied upon by either side, are
discussed later in my assessment of their evidence.
Environmental experts
[23]
Three environmental expert witnesses were led for the pursuers (Mr Brien, Mr Quint
and Mr Witherington) and one expert gave evidence for the defender (Mr Firth). The central
themes were looking at the types of chemicals, solvents and contaminants previously found
on the site and in particular areas, and reaching a view on the level of contamination likely
to have been caused. This was done principally by reviewing the historical environmental
reports of WSP, Monridge and IKM and commenting on the figures identified in those
investigations. Accordingly, these experts did not, for the purpose of their reports, carry out
their own sampling.
Pursuers' environmental experts
Mr Brien
[24]
Kevin Brien is an experienced professional environmental engineer, licensed in
Pennsylvania and Delaware. In his expert report, Mr Brien gave his opinion on the reasons
for the differences in results obtained by WSP, Monridge and IKM in relation to soil
sampling, soil vapour and indoor air quality sampling. He commented on concerns raised
by Environ, instructed on behalf of the defender, about work completed by him when he ran
Monridge. Mr Brien then gave his own views on the extent of contamination in soil, soil
vapour, and groundwater at the site and on the shortcomings of conceptual models used by
12
consultants, such as WSP, to evaluate risk. In relation to soil contamination, his view is that
the groundworks have spread contaminants throughout the site. On soil vapour, his
opinion is that the extent of soil vapour impacts has not been properly investigated. He also
comments on the soil probes installed by WSP, saying these were not appropriate for
assessing the risk associated with soil vapour throughout the site, a point now agreed
between the experts
[25]
In his opinion, the horizontal and vertical delineation of soil, soil vapour and
groundwater at the site is incomplete and the methodologies used and errors in the
investigative approach have resulted in a dataset that cannot be relied upon to properly
assess the risk to residents at the development. This applied to WSP but also to the
analytical models in the Quint and Firth reports.
Mr Quint
[26]
Michael Quint is an expert on contaminated land risk assessment. He concluded that
Mr Firth's report does not represent a full land-contamination risk assessment of the site. It
is restricted to a consideration of vapour risks from a subset of the detected subsurface
contaminants. Other issues relevant to the pursuers' claims, such as potential direct contact
with contamination (soil ingestion and dermal absorption), have not been considered by
Mr Firth. Mr Quint refers to significant limitations with the available site investigation data,
and states that these have not been sufficiently recognised. While he generally agreed with
Mr Firth's final opinions regarding the questions he was asked, Mr Quint felt that Mr Firth's
modelling of potential indoor vapour levels in the pursuers' homes is not as health-
conservative as it should have been. Taken together, the various sources of uncertainty
make the results indicative only, such that they could represent over- or under-estimates of
13
the levels of risks encountered by the pursuers. Mr Quint also carried out his own vapour
modelling exercise.
[27]
He concluded that, on the basis of his updated human health risk assessment, there
appear to be, and will have been historically, human health risks associated with
contamination at the Watling Street site, extending over a number of years, since the
tenancies started, and continuing to the present day. For him, this is supported by the
following: measured levels of chemicals in soil, at certain locations, exceeding GACs and
site-specific assessment criteria; measured levels of chemicals in soil, soil vapour and
groundwater having been shown, by computer modelling, to be capable of causing indoor
air concentrations in excess of health-based assessment values (HBAVs); chemicals
associated with site-related contamination having been measured in indoor air in excess of
HBAVs; and chemicals associated with site-related contamination having been detected in
outdoor air.
[28]
Further sources of contamination risk could, in his opinion, also exist at the site.
These are undetected, due to important limitations with regard to the environmental
sampling which has taken place there, to date. In his view, the identified contamination has
resulted in the properties not being capable of occupation for a reasonable time without risk
to the health or safety of the occupants, although whether there has been any actual harm to
human health is beyond the scope of his expertise, and he defers to others. However, he
also states that "the true risks will never be known, and they could be greater than those
implied here, or by Mr Firth, IKM, or WSP, or they could be zero." In re-examination,
Mr Quint stated that the intention of his work was to provide insight into what risks
"theoretically there might have been over the years".
14
Mr Witherington
[29]
Peter Witherington is an expert in geotechnical and environmental land related
issues. On the basis of the information available, he concluded that there are two potential
causes for contamination being present in the ground around properties across the site. The
first is that appropriate contaminants were not tested for during the investigation phases of
works and as a result the presence of residual VOCs in the ground was not considered.
These substances could then pose a potential risk to the future residential properties "via
direct contact exposure to shallow soils and via vapour intrusion and indoor inhalation
exposure by residents". These substances have been identified in subsequent investigations
carried out by WSP but he considers the sampling methods used could have resulted in
under-recording of the concentrations. Furthermore, because of volatilisation and other
degradation processes, concentrations may well have been higher at the time the
development was completed.
[30]
Secondly, across most of the site the requirement for a suitable thickness of
subsoil/topsoil layer in garden areas, as recommended by SWK, appears to have been
fulfilled using site-won clay containing varying quantities of ash, slag, clinker and other
anthropogenic materials. This is particularly evident in the even-numbered northern
properties on Forum Place. This has resulted in those hotspots identified by WSP, which
they recommended be removed.
[31]
In his opinion, residents at the site are more likely than not to have been exposed to
higher levels of VOCs in their gardens than they would have otherwise been exposed to if
adequate investigations had been undertaken prior to development and appropriate
remedial works taken to resolve them.
15
Defender's environmental expert
Mr Firth
[32]
Simon Firth is a contaminated land practitioner who specialises in human health and
environmental risk assessment of subsurface contamination. He was instructed for the
defender to prepare an expert technical report in relation to vapour risks from subsurface
contamination. In his opinion, on the balance of probabilities, a viable pathway exists by
which vapours (from the subsurface) could enter the properties and vapour intrusion has
been ongoing since the buildings were constructed.
[33]
Mr Firth dealt specifically with vapour modelling, which he conducted to predict the
concentrations of vapours in indoor air arising from the measured concentrations in soil, soil
vapour and groundwater. His conclusions were that the predicted indoor air concentrations
based on the measured soil concentrations are all below health-based screening criteria, with
certain exceptions. There was disparity between the WSP results and the Monridge results,
but doubt was cast on the validity of the Monridge dataset and it is possible that the indoor
air concentrations of carbon tetrachloride have been over-predicted by Monridge.
[34]
The indoor air concentrations predicted from the measured WSP sub-slab
concentrations are all below current health-based screening concentrations. The indoor air
concentrations predicted from the measured Monridge soil vapour concentrations exceed
those screening concentrations for long-term exposure for TCE and carbon tetrachloride.
However, the predicted concentration of TCE in indoor air (9 g.m-3) is below the previous
screening value of 18.2 g.m-3 which would have been applicable at the commencement of
the pursuers' tenancies. Furthermore, carbon tetrachloride was not detected by Monridge in
indoor air samples. Groundwater vapour modelling has shown that volatilisation from
groundwater is unlikely to result in indoor air concentrations exceeding health-based
16
screening values. Monridge used what were called "grab samples" and more detailed
investigations were made by WSP.
[35]
In relation to the gardens, the type of vapours present in outdoor air, arising from
subsurface contamination, are likely to be the same as in indoor air arising from subsurface
contamination. However, the concentrations of vapours in outdoor air are predicted to be
several orders of magnitude below those in indoor air and are well below screening values
that represent low/tolerable risk for long term (chronic) exposure.
Toxicology experts
[36]
Expert evidence on toxicology, that is, whether chemicals or substances are toxic or
harmful to humans, was given for the pursuers by Professor Douglas and for the defender
by Professor Middleton.
Professor Douglas
[37]
In considering the evidence of Professor Douglas, it is appropriate to note that he
agreed with a number of points made by Dr Bojan Flaks, an expert previously instructed for
the pursuers who is now deceased. In the opinion of Dr Flaks, it appeared that housing was
built on this former industrial estate without adequate prior remediation. Environmental
investigations, notably by Monridge and by IKM, have in his view shown that the ground of
the site is contaminated with a variety of toxic organic solvents.
[38]
The final conclusions reached by Dr Flaks can be summarised as follows. On the
basis of the results of the environmental, structural and health investigations that have been
carried out: if toxic solvents at the level detected by the testing that has been done are
present in the soil in the vicinity of the house and there is a pathway that permits the
17
migration of the solvent into the house, and the solvents are present in the indoor air at the
levels indicated by the investigations that have been carried out, then, on the balance of
probabilities, the residents of that house will have suffered from some form of neurotoxic or
other damage. For any particular individual it would be necessary to exclude alternative
causes, at least by review of his or her medical history.
[39]
Professor Douglas was asked to prepare an independent critical review of the report
of Dr Flaks. He noted Dr Flaks as stating that it appears that solvents are able to enter the
residential premises readily as vapours by diffusion, thereby exposing the occupants, and
that there is also the potential for a dermal pathway from the contamination in the gardens.
Professor Douglas mentions the reference by Dr Flaks to the Motherwell Health Survey from
May/June 2010, a symptom questionnaire based on 99 individuals at the site. He notes that
there are some clearly high values for anxiety (18%), depression (20%) and, particularly,
headaches (41%), asthma (12%), other respiratory problems (15%) and that there are also
some interesting low values, including stress (1%), poor concentration (1%) and disturbed
sleep (1%).
[40]
Professor Douglas describes two matters in Dr Flaks' report as cardinal points:
firstly, that the soil contains a list of solvents at high levels (including vinyl chloride, among
others); secondly, that migration of solvents into the houses is possible. He concludes that
Dr Flaks has produced a thorough, comprehensive toxicological report into solvent
contamination at the site. He says that, assuming Dr Flaks has accurately described the
factual background and accurately summarised the investigations mentioned, he agrees
with the conclusions Dr Flaks has reached. Professor Douglas also observed that as well as
what Dr Flaks referred to about people having suffered symptoms, others in the future
would also suffer.
18
[41]
He goes on to discuss in some detail the risk of cancer. He concludes in his report
that at the commencement of the tenancies, on the basis of the factual material described and
Dr Flak's report, on the balance of probabilities, the housing association was in breach of its
statutory duty.
[42]
Professor Douglas then reviewed the witness statements of the residents. He
mentions that some appear to have shown a degree of recovery after removal and others
not. He comments on social circumstances of many of the residents and talks about
historical studies showing levels of anxiety among persons living in deprived circumstances
in Glasgow and low life-expectancy of working men. He says that thus, when moving
further into the records provided, it is no great surprise to find extensive medical records,
reflecting ill-health and adverse social circumstances as well as housing worries. He
observes, as a common thread, that those person who have moved out of the properties have
found an improvement in their health whilst others have noted the worsening of their health
after moving in, only to resolve on moving out. He also recognises, given the amount of
documentation and medical records, that it will be difficult to sort out for each individual
the potential sources of ill-health, reflected in large numbers of GP visits and large quantities
of medicines prescribed.
[43]
He concludes that if the court accepts that environmental pollution in the form of
heavy metals and solvents was present then, on the balance of probabilities, this is likely to
have contributed to the ill-health of the residents, additional to that determined by the
socioeconomic status of many of the residents on the estate.
19
Professor Eddleston
[44]
Professor Eddleston is a Professor of Clinical Toxicology at the University of
Edinburgh and a consultant clinical toxicologist and pharmacologist. Having regard to the
modelling of Simon Firth, he reached the following conclusions. On the balance of
probabilities, there is no evidence from the medical records that any of the exceedances
noted in Mr Firth's modelling gave rise to symptoms or harms. He refers to the pursuers'
pleaded position on the workplace limits for contamination, including that chronic solvent
exposure is well-recognised as being injurious to health in the workplace, and there could be
harm caused by chronic exposure of occupants of residential properties at lower
concentrations than has been set for workplace exposure. In his opinion, this is a theoretical
position. Workplace exposure limits are set conservatively, usually 10-100 times lower than
the calculated dose at which no adverse effects are observed. He was unable to find any
cases in the literature where people were potentially exposed to solvent levels below the
occupational or workplace limits and developed clear toxicity specific to the compounds
involved, or indeed developed any disease.
[45]
He was asked four questions by the instructing solicitors for the defender, based on
the assumption that he is content that, if the Firth modelling is reliable, the only concerns are
with 25 and 29 Empire Way and 2 Tiber Avenue on the potential risk to human health. The
first question was about the levels of TCE and carbon tetrachloride from which one would
expect symptoms to be present, and the symptoms that might arise. His response was that
he would expect symptoms if air concentrations exceeded the long-term UK workplace
exposure limits (over 8 hours) for TCE (100 ppm, 550,000 g/m3) or carbon tetrachloride
(1 ppm, 6,400 g/m3). No exposure limits have been developed for longer duration
20
exposure. He felt that it is important to remember that exposure limits are set to avoid any
ill-health in workers and so have a large safety margin.
[46]
The second question was whether, on the balance of probabilities, the levels of TCE
and carbon tetrachloride estimated above could give rise to the symptoms complained of by
the tenants at the three properties he was asked to consider. His answer was, on the balance
of probabilities, the modelled concentrations of TCE and carbon tetrachloride in the
properties would not explain the symptoms reported. The workplace exposure limits
(concentrations) are substantially higher than the values modelled in the Firth report
(TCE: 4.9 g/m3; carbon tetrachloride 14-27 g/m3). This marked difference between
modelled concentrations and workplace limits is described by him as reassuring.
[47]
The third question concerned the extent to which, if any, the findings of the report by
Mr Firth alter his own conclusions in relation to the tenants at these three properties. His
response was that the report's findings did not alter his conclusions.
[48]
The final question was whether there were any other findings in the Firth report
which Professor Eddleston thinks may be relevant to the question of whether the vapours
present in the properties could have resulted in the health effects alleged by the pursuers.
He did not consider any other findings in the Firth report to be relevant to this question.
[49]
Professor Eddleston noted that there are certain symptoms associated with what is
known about exposure to solvents, but he pointed to the absence of research which
establishes that there is a link between these symptoms and chronic low-level exposure. He
also points out that many of the symptoms that are complained about are symptoms that
people commonly suffer from, and that many of the pursuers, but not all of them, had been
recorded as experiencing those types of symptoms before their tenancies began. He notes
that the medical records of many of the pursuers did not record those complaints at the time.
21
[50]
He stated in evidence that what is recorded in the medical records can be treated as a
reliable account of the symptoms that people were suffering from at the time. His view was
that the GP would likely have been told of all the material problems that the individual was
suffering from when visiting the GP, and the GP would have recorded them. In response to
a question that in the course of an assessment he cannot purely refer to GP records and
ignore what the patients themselves have described, he said that all people tell stories about
when they talked to doctors, whereas GP records are objective and are a concurrent or
contemporaneous report. There would be no reason to think that a person would not have
reported something that is viewed as particularly bad, because a person who is unwell
would generally report all of the symptoms to the GP.
[51]
Professor Eddleston summarised his position. Mr Firth's report, describing his
modelling of potential in-house exposures to VOCs, reported four exceedances of the
current long-term occupational concentrations. Review of the medical records for the
inhabitants of the houses involved found no evidence to indicate any illness reported to be
associated with the relevant chemicals at higher concentrations. On the balance of
probabilities, in the absence of any supporting information, the illnesses reported by the
pursuers did not result from low-level in-house exposure to VOCs in the Watling Street
development.
Submissions
[52]
The statutory provisions noted above were first introduced in similar terms by the
Housing of the Working Classes Act 1890. Parties each accepted the interpretation given in
certain key cases. These include the English authorities Morgan v Liverpool Corporation
[1927] 2 KB 131 and Summers v Salford Corporation [1943] AC 283, the reasoning in these cases
22
having been followed in several cases in Scotland. Each counsel also referred to what they
described as analogous statutory provisions, which I shall discuss briefly when I come to
explain my decision and reasons.
[53]
Counsel prepared written submissions and these were then developed in oral
submissions, responding to the other side's position and offering criticisms or challenges. I
have considered all of the submissions. When I come to assess the evidence I will deal with
the relevant points in contention and at this stage it suffices to summarise the main points
made by each side.
Submissions for the pursuers
[54]
The full extent of contamination at any individual property (including the garden) is
not known, but it has been there since the commencement of each tenancy. There could be
other contaminants present within Plot A which were not analysed within the course of the
WSP site investigations. WSP's methodology and sampling were not sufficient, including
the depths at which sampling was taken. Vapour inhalation from the contaminants is
considered to be a significant potential risk. Mr Brien and Mr Quint both concluded that
there is a risk of undiscovered hotspots of contaminants at the site. There is no record and
no evidence that the identified hotspots were excavated and disposed off-site and replaced
by clean fill soil. As Mr Brien stated, there are likely to be more hotspots and contaminants
at the site and the contaminants concerned are more than just the TCE which Mr Firth was
focusing on. What was found elsewhere could be significant in relation to what could
potentially be found at a pursuer's property if further investigation was undertaken. From
WSP's methodology there isn't a clear understanding of the situation regarding
groundwater.
23
[55]
Mr Quint's modelling method was more appropriate than that of Mr Firth. In any
event the modelling carried out by Mr Quint and Mr Firth is of limited value in this case.
The exercise of modelling is based on estimates and assumptions and is not a science. It
post-dates the commencement date of most of the tenancies and deals only with a single
contaminant at a time. The data is not reliable to allow meaningful modelling. The
modelling does not help in knowing the full range of potential contamination exposure at
the site generally or at individual properties. It deals with the consideration of vapour
concentrations found in the particular examples in comparison with GACs used in relation
to site development. Part 2A of the Environmental Protection Act 1990, on which the
defender places some reliance, is not relevant and it deals with different issues. Moreover,
the GACs for exposure to TCE have been lowered, indicating the knowledge of a higher
health risk. It is also necessary to take into account the carcogenicity of TCE.
[56]
The pursuers recognise there is a lack of research to show what the effects of chronic
exposure to low-level solvent fumes might be. The pursuers also recognise that it is not
possible to know with any certainty what the concentration of exposure to any particular
contaminant might have been. However, there are four points which show the impact on
the pursuers' health. Firstly, their own individual reported experiences concerning the state
of their health before they moved into the tenancy, whilst they were living there and the
differences, in symptoms and health, after they moved out. Secondly, they show a collective
experience of their health problems and the similarity of the symptoms. Thirdly, they
mention the experience of others who lived in the house or visited the house. Fourthly, for
some of the pursuers there is stronger evidence of linkage to potentially harmful levels of
contaminants in the ground than for others.
24
[57]
On that last point, WSP had identified hotspots at 18 Forum Place and 2 Tiber
Avenue. On the balance of probabilities it is likely that they remain in place. The property
at 31 Empire Way is a ground floor property, and 29 Empire Way is a first-floor property.
Vapour fumes present in 31 Empire Way are also likely to permeate 29 Empire Way, so the
findings in relation to 31 Empire Way are also indicative of contamination from the same
solvent vapour fumes as 29 Empire Way. Also, certain witness suffered rashes after having
planted flowers or done other gardening activities. Given the evidence of Mr Witherington
and Mr Brien that contaminants are all over the site, and the figures provided by Mr Firth,
on the balance of probabilities these rashes are caused by direct or dermal contact with
contaminants in the ground.
[58]
Professor Eddleston noted that there has never been a community affected like this,
according to the literature. He does not, however, attempt to consider any commonality
between the pursuers' symptoms and what that commonality indicates. He views the
pursuers as not credible or reliable. Professor Eddleston attributed the linking of their
health problems to the occupation of their housing as being due to health anxiety, arising
from being told about contamination. However, he did not explain why tenants who had
left their houses reported feeling better before the contamination of the site became common
knowledge.
[59]
Professor Eddleston did not consider certain relevant matters and speculated about
alternative explanations. He did not take into account the potential for synergy,
additionality or compound effects due to the risk of there being more than one source of
contamination exposure in the properties. Professor Douglas, an expert with knowledge
and experience, gave an illustration of the impact of smoking and exposure to asbestos being
multiplicative. In addition to considering the medical information of each individual he
25
looked at the pursuers as a group and identified commonalities. He considered the overall
impact of exposure upon the pursuers' health and whether it caused or exacerbated
symptoms.
[60]
The risk of harm requires to be considered by reference to the agreed pathways
linking contamination with the pursuers, including direct contact with soil or the presence
of hotspots not shown to have been removed. In relation to vapour inhalation, on the
balance of probabilities the ground on which the homes and gardens have been built do
contain VOCs and SVOCs. The pursuers' evidence should be accepted, showing that they
have experienced harm from the contamination of the property while they have been
residents.
[61]
Professor Douglas`s evidence and his support of the pursuers should be preferred to
Professor Eddleston's evidence to the contrary. Professor Eddleston accepted that the
ailments and symptoms could potentially have been caused by vapour inhalation and his
issue is simply that regarding chronic exposure to multiple contaminants there is no medical
evidence of a link. Professor Douglas, based on his experience, says there would be a link by
analogy with other situations.
[62]
The real issue is actual or potential harm to health. An increased risk of cancer is
something which you can get damages for and you do not have to get cancer to suffer. On
the evidence, it has been demonstrated that there was a risk to health and indeed that is
referenced by Mr Firth in his report. In relation to petroleum hydrocarbons being found, it
was not a credible proposition that petroleum had been brought inside the premises by the
tenants. The defender's point about commonality being restricted as there are many other
residents (some 200 in total) is of no significance as we do not know what symptoms the
others have also suffered and there are also other litigations going on.
26
Submissions for the defender
[63]
The point that there could be other contaminants on the site is not enough for the
pursuers to prove breach of duty. It is for the pursuers to show that there are in fact other
contaminants present. Also, when a case is based on harm at individual properties it would
not only be useful to know, but an important thing to prove, the extent of contamination at
an individual property. It is accepted that in principle the presence of contamination could
be capable of rendering the subject properties unfit for human habitation. However, Part 2A
of the 1990 Act should be considered and indeed it would be anomalous if the court were to
hold that subjects which do not meet the threshold for contaminated land under the
1990 Act are unfit for human habitation under and in terms of the 1987 or 2001 Act.
[64]
In relation to factual evidence, it is accepted that the individual pursuers were
generally doing their best to assist the court in their witness statements and oral evidence.
However, the evidence of Professor Eddleston about the pursuers' accounts of their ailments
and symptoms is relied upon.
[65]
On the environmental evidence of Mr Brien, Mr Witherington and Mr Quint the
opinion evidence they gave does not support a conclusion, on the balance of probabilities,
that the properties are not reasonably fit for human habitation or that there is any risk to the
health of the occupants. Taken at its highest, the evidence is only capable of supporting the
proposition that further investigations would be required before any conclusions could be
drawn regarding those matters. Mr Brien took the view that investigations were incomplete
and the dataset cannot be relied upon to properly assess the risk to residents.
Mr Witherington was not asked to address the question of risk. Mr Quint commented on
27
modelling and various sources of uncertainty making things incomplete and said the true
risk was not known.
[66]
MTBE was not detected in any of the soil samples, or soil vapour samples, of which
there were over 200 taken across the site and there are no other samples exposing MTBE.
This suggests that if it is in indoor air it is not coming from the ground. In relation to
groundwater, WSP found that the granular horizon was not laterally continuous and
shallow groundwater was likely to be discontinuous. In other words, there was no pathway
for groundwater to move around the site. On the point raised about the carcogenicity of
TCE, that is just another mention of a potential risk rather than an actual risk.
[67]
On the hotspot point, Mr Firth gave coherent reasons to support his conclusion that
there are unlikely to be any further hotspots at the site. There is no proper evidential basis
from which to conclude that further hotspots exist. Even if undiscovered hotspots do exist,
Mr Brien accepted that he cannot say where they would be and which of the pursuers, if
any, they may affect. While the pursuers say that there is no record and no evidence that the
identified hotspots were excavated and disposed off-site and replaced by clean fill soil, there
is no evidence that they were not excavated and disposed.
[68]
In relation to modelling, notwithstanding the uncertainties and data gaps there is
sufficient data available to conduct meaningful analytical modelling. Mr Firth's approach
should be preferred. He has conducted vapour modelling for the main VOC solvents
detected at the site and the thresholds identified represent a minimal or low risk to health.
[69]
Mr Quint's approach was not consistent with the pursuers having the burden of
proof on the balance of probabilities. However, even if the figures from Mr Quint's
modelling were to be adopted, the pursuers have not led evidence which would allow the
28
court to conclude that the properties were not reasonably fit for human habitation or that the
pursuers suffered ill-health due to the presence of contamination at the site.
[70]
In terms of the expert medical evidence, there are a number of problems with the
evidence of Professor Douglas, including that he is not a clinician and has no experience of
diagnosing patients. Professor Douglas repeatedly stated that he had focused on the
pursuers' witness statements and did not appear to attach much significance to the medical
records, which Professor Eddleston relied upon for the reasons given earlier.
[71]
Professor Douglas's conclusions are not properly supported. His expert evidence
fails to satisfy the criteria discussed in Kennedy v Cordia (Services) LLP 2016 SC (UKSC) 59.
[72]
The pursuers say it is not possible to know with any certainty what exposure to
contaminants might be and might have been at commencement. The pursuers make no
attempt to draw any conclusions about likely levels of exposure. While occupational or
workplace exposure is not directly relevant, there is an enormous disparity and several
orders of magnitude between what was found here and what is referred to in the guidance
on occupational exposure.
[73]
Thirteen individual pursuers are not particularly significant when there could be
200 residents at the site. Dealing with a self-collective group of people who have chosen to
raise proceedings is inappropriate. As to the experience of visitors, we do not have the
medical records and they didn't give evidence. No weight can be placed on that matter.
[74]
Professor Douglas does not offer any basis as to why rashes could be caused by
personal contact. What the pursuers said about garden soil and not growing plants does not
assist. The evidence does not come close to establishing any causation on the balance of
probabilities. There is no information, in relation to the various other litigations referred to
by Mr Sutherland, of residents or what was complained of by them.
29
Decision and reasons
Relevant law
[75]
As noted earlier, each counsel referred to what they described as analogous statutory
provisions to those in the 1987 Act and the 2001 Act. For the pursuers, these were
section 2(1) of the Occupiers Liability (Scotland) Act 1960, dealing with a tenantable
condition and section 1 of the Defective Premises Act 1972, about premises being fit for
habitation. Mr Sutherland referred to case law, including an English case on the 1972 Act
about risk to the health or safety of the occupants.
[76]
For the defender, reference was made to Part 2A of the Environmental Protection
Act 1990. This piece of legislation seeks to enable the identification and remediation of land
in which contamination currently represents an unacceptable risk to human health.
Contaminated land is defined as land which, by reason of substances in or under the land,
significant harm is being caused or there is a significant possibility of such harm being
caused.
[77]
While some degree of analogous wording is present in these other statutes, the
language used in the provisions is not identical to that in the 1987 Act and the 2001 Act (for
example, there are the references to "significant" in the 1990 Act). The appropriate course is
to apply the terms of the 1987 Act and the 2001 Act, as construed in the authorities.
[78]
As to the case law on that legislation, in the leading authority, Morgan v Liverpool
Corporation, it is stated (at 145) that:
"If the state of repair of a house is such that by ordinary user damage may naturally
be caused to the occupier, either in respect of personal injury to life or limb or injury
to health, then the house is not in all respects reasonably fit for human habitation."
30
This accurately represents the interpretation of the relevant provisions. The approach in
Morgan is reflected in other decisions, such as Summers v Salford Corporation and in Scottish
cases such as Haggarty (No 2) v Glasgow Corporation 1964 SLT (Notes) 54, Christian v Aberdeen
City Council 2006 SCLR 448 and Todd v Clapperton 2009 SLT 837. For breach of duty, it is
therefore for the pursuers to prove, on the balance of probabilities, that the test in Morgan is
met.
[79]
The standard approach to proof of causation is that a pursuer needs firstly to
demonstrate, on the balance of probabilities, that the event which allegedly gave rise to the
pursuer's damage can ever cause that type of harm. Secondly, once it is established that the
event can cause such damage the pursuer requires to prove, at the same standard, that the
particular damage was caused that way (see eg Clerk & Lindsell on Torts, 24th ed, at paras 2-28
to 2-31). Illustrating the first point, in Kay's Tutor v Ayrshire and Arran Health Board 1987 SC
(HL) 145, the House of Lords held that the law could not presume that the breach of duty
was responsible if it was not proved, or an accepted fact, that the breach was capable of
causing or aggravating such damage. Lord Keith referred to the absence of acceptable
medical evidence on that matter.
[80]
If the first point (capable of causing) is proved that can in some cases allow the
second point (actually causing) to be presumed or inferred. For example, it is possible for a
material increase in risk to allow the inference that it did cause or materially contribute to
the harm (McGhee v National Coal Board [1973] 1 WLR 1). However, that was said in
circumstances in which it was established that the exposure (in that case, to brick dust)
undoubtedly caused such harm; it was a known factor that would lead to the injury and the
material increase in risk resulted in the inference that it had done so.
31
[81]
If there can be other factors which cause the harm, it would have to be shown that
the harm was actually caused, or materially contributed to, by the factor concerned
(Wilsher v Essex AHA [1988] AC 1074). That is also illustrated in Petroleum Co of Trinidad and
Tobago Ltd v Ryan [2017] UKPC 30, which shows that where there are a number of possible
causes of a disease or disability, one of which is capable of being the defender's breach of
duty, the onus remains on the pursuer to show that the breach made at least a material
contribution.
[82]
As there are multiple potential causes of the symptoms of the pursuers, proof that a
breach of duty is capable of causing them will not suffice to show it did cause them.
However, if the pursuers prove that some or all of the symptoms suffered by them were in
fact caused by the ordinary exposure to contaminants on the site, then the point about the
breach being capable of causing them is obviously also established. Indeed, given the nature
of the statutory duty, proof of actual cause of the harm will suffice for both breach of duty
and causation. In reaching my views on these issues, it is appropriate to assess the two
types of expert evidence in turn, taking into account all of the factual evidence.
Assessment of environmental experts' evidence
Part 2A of the 1990 Act
[83]
The threshold for "contaminated land" under Part 2A will not determine whether or
not there is a breach of duty. It is, however, referred to by various experts and in the parties'
submissions. A brief overview of Part 2A is necessary to understand the evidence. Part 2A
involves determination of the level of contamination. Statutory guidance, which applies in
England, Wales and Northern Ireland, introduced a four-category system for classifying
land under Part 2A to identify a Significant Possibility of Significant Harm (SPOSH) to
32
human health. It is a precautionary approach. In relation to the four-category system, land
is determined as contaminated land under Part 2A if it falls within Categories 1 or 2. If the
results are at, or above, the screening level for Category 1, then the level of risk is clearly
unacceptable.
[84]
The Department of Environment, Food and Rural Affairs (DEFRA) produced a table
in 2011 which sets out the GACs and SGVs and Category 4 screening levels (C4SLs). If the
results are below the Category 4 screening level, then the level of contamination is
acceptably low. Category 4 provides a simple test for deciding when land is suitable for use
and definitely not contaminated land. Such low-risk land can then be dismissed from the
need for further risk assessment. In the very large majority of cases, the GACs and SGVs
describe levels of contamination from which risks should be considered to be comfortably
within Category 4. They are designed to estimate levels of contamination at which risks are
likely to be negligible or minimal.
[85]
So, these tests deal with the question of whether there is a risk to human health,
albeit for the purposes of checking contaminated land in terms of Part 2A. Nonetheless, it is
useful to know which Categories, 1 to 4, the risks in the present case fall within. While
falling below Category 4 does not necessarily mean that there is no risk at all to health, it
does point in that direction and generally does so even more strongly if the level is below
the GACs or SGVs. It would, therefore, be inappropriate to reject the modelling data merely
because it is linked to Part 2A.
Methods of modelling
[86]
Mr Quint explained that the true mean at a contaminated site is normally unknown,
given the amount of soil testing that would be involved in establishing it. It is impractical to
33
have hundreds of thousands of samples and the issue is therefore how to properly estimate
the true mean. No specific guidance exists on how land contamination should be assessed
in terms of whether a property is reasonably fit for human habitation. However, as
Mr Quint explained, extensive government guidance, including that referred to above, is
available for land contamination risk assessment in other circumstances.
[87]
In seeking to determine the true mean for the levels of contaminants, Mr Firth and
Mr Quint agreed that CLEA (Environmental Agency's Contaminated Land Exposure
Assessment) and J & E (US Environmental Protection Agency's Johnson and Ettinger)
models are appropriate to use for the vapour modelling assessment. The experts each
considered the results from the investigations conducted by WSP, Monridge and IKM,
discounting the WSP vapour probes.
[88]
For the modelling, which seeks to identify the long-term average concentration of
VOCs in air, they agreed on what were the key parameters. But in relation to vapour
intrusion from soil concentrations they took different approaches. Their different
parameters led to significantly different results. Mr Quint applied a higher test than
Mr Firth, using risk analysis methods which introduce a protective or health-conservative
approach. This involved what is called a 95% Upper Confidence Limit (UCL95).
[89]
Using UCL95 does not seek to identify the most likely average, or mean, figure for
the level of contaminant. Rather, it generates a figure which gives 95% confidence that the
true average concentration will be less than or equal to that calculated figure. As Mr Quint
put it, if a sampling exercise was repeated 20 times, on 19 occasions the calculated sample
mean would be less than, or equal to, the UCL95 value. The details of this method of
calculation do not need to be explained further, but it is a conservative approach, creating a
figure which gives that very high level of confidence. Mr Quint took the view that using
34
UCL95, rather than a straightforward average, is appropriate for this assessment, given the
data uncertainty and the fact that the data was collected at least 10 years after the site was
first occupied for residential use.
[90]
Mr Firth used the arithmetical mean figure. He did so because the purpose of
analysis in the present case is about whether, on the balance of probabilities, there was a
breach of duty by the defender. There was a relatively small amount of data and in his view
this resulted in the calculations by Mr Quint, using the UCL95 approach, generating higher
figures for risk assessment. Mr Firth gave an example in his evidence of the effect on results
of the different approach taken by Mr Quint. At 25 Empire Way, Mr Firth was predicting, in
relation to soil concentration, a figure of 4.2 g.m-3 for TCE, whereas Mr Quint was
predicting some five times higher than that, having only changed two parameters. Mr Firth
described his own approach as being to use the most likely parameter values to get the most
likely indoor air concentration. For him, this was more appropriate than being cautious and
using a value that over-predicts the level of risk. He was looking for the most likely results
rather than using figures skewed, out of caution, at a higher level.
[91]
Part of Mr Quint's justification for his approach, that the concentrations of the
relevant contaminants were higher at the start of the tenancies than at the date when the
modelling is based, is not well-founded. The tenancies in question started on a range of
dates (from December 1999 to October 2011) with roughly half of them starting from 2008 or
later. So the assumed time gap between the start of the tenancy and the date of sampling is
immaterial for a number of the pursuers. Moreover, it was not possible for the experts to
say which concentrations will have reduced over time, with Mr Firth saying that any year-
to-year concentration reductions is likely to be modest and that VOCs will take longer to
break down in shallow soils.
35
[92]
As a result, I am not persuaded that Mr Quint's more conservative approach is
justified. In my view, Mr Firth took a more objective approach to modelling in light of the
central point about whether or not there is a breach of duty. Mr Quint's conservative
approach is more likely to result in an over-estimation of indoor air concentrations than
Mr Firth's being an under-estimation.
[93]
There is some room for concern about whether there is sufficient data to allow these
modelling exercises to properly assist in determining the issues here. But as the experts
agreed, there are key limitations and data gaps in the information available and in those
circumstances having results from modelling can provide a degree of assistance in working
out the level of risk. It is used to predict long-term average concentrations of the
constituents of potential concern in indoor and outdoor air, from soil and groundwater,
respectively. If the modelling had not been used at all there would have been even less
information to consider in understanding the long-term levels of contamination. Analytical
modelling is used on that basis and provides some meaningful information.
[94]
All of that said, perhaps the key point to be taken from the evidence of the
environmental experts is that even if the figures from Mr Quint's modelling were to be
accepted, the pursuers still need to lead sufficient evidence which would allow the court to
conclude that the properties were not reasonably fit for human habitation. As noted earlier,
Mr Quint said in evidence that the true risks will never be known, his work being to provide
insight into what risks "theoretically there might have been over the years". If that central
requirement of sufficient evidence is not met, the basis for the modelling and the effect of its
results are, in effect, academic.
36
Contaminants detected above screening levels, GACs or SGVs
[95]
As already explained, the screening levels, while not directly applicable to the
statutory test in this case, are used to show that long-term human health risks for site
occupants are considered to be tolerable or minimal. Where a screening level, GAC or SGV
is exceeded, the expert evidence is broadly to the effect that this does not necessarily
correspond to a risk of harm to human health. GACs and SGVs are considered to be
conservative criteria and results above those levels simply mean that further consideration is
required, rather than that a risk of harm has occurred. The purpose of the screening levels,
GACs or SGVs, is therefore to screen out contaminants from more detailed assessment, as
explained in the Society of Brownfield Risk Assessment (SoBRA) guidance and in the
DEFRA statutory guidance. Exceedance merely means that further assessment is needed
before a proper conclusion can be reached. The existence of examples exceeding the levels
does not therefore prove the case for the pursuers.
Hotspots and undiscovered hotspots
[96]
The fundamental thrust of the pursuers' expert evidence on hotspots is the likelihood
that there are other undiscovered hotspots on the site which might then cause a level of risk
to occupants. The hotspots identified at, or near to, particular locations were found several
years after certain tenants moved in. Mr Brien, for Monridge, dug at a deeper level at
31 Empire Way and found higher levels of contaminants.
[97]
Mr Firth gave coherent and convincing reasons to support his conclusion that there
are unlikely to be any further hotspots at the site. These included that the spatial
distribution of the sampling done by WSP correlated with the spatial distribution of TCE
concentrations in soil. In other words, most of the areas looked at were found to be at a low-
37
level of contaminants. A number of boreholes were taken across the site, at a reasonable
depth, and no further hotspots have been identified. Also, brick structures present at the
north west of Forum Place made it reasonable to conclude that the TCE hotspot there is
likely to be associated with those bricks.
[98]
I do not consider that Mr Brien or Mr Quint are able to properly infer a likelihood of
further hotspots on a sound and persuasive basis, when there is no proper evidential basis
for saying they exist. Moreover there is nothing to indicate where any such undiscovered
hotspots would be located and therefore whether they could have affected any of the
pursuers. The fact that hotspots were present when certain tenants moved in is a factor to
consider, but the overall position of the pursuers' evidence is that they all suffered common
symptoms, even those not living beside identified hotspots. The absence of evidence of
hotspots being excavated and removed does not take us any further, because there is also no
evidence showing or suggesting this was not done.
PAHs
[99]
WSP's assessment was that two gardens (including 2 Tiber Avenue) had a hotspot of
polycyclic aromatic hydrocarbon (PAH) concentrations above residential screening criteria
and another (18 Forum Place) had two such hotspots. Monridge testing also detected
concentrations above the level. There is no dispute that PAHs in direct contact with soil is a
linkage of potential concern, as the detection limit exceeds GACs. However, as already
noted, exceeding GACs does not of itself demonstrate risk, and requires further
investigation. Mr Firth concluded that, based on Mr Quint's and WSP's risk assessments
and with the possible exception of 2 Tiber Avenue and 18 Forum Place, non-vapour
pathways are not of concern for any of the other properties on the site. Based on his
38
reasoning and analysis, including about the precautionary approach taken by Mr Quint in
his modelling and Mr Firth's observations about the Monridge assessments, I accept that
view. As a result, the possible exceptions remain of some potential concern, but there is no
evidence taking the concern beyond that level.
Petroleum hydrocarbons
[100]
Petroleum contaminants (such as benzene, toluene, ethylbenzene, xylene) have been
detected by WSP in areas around and under 13 Tiber Avenue and 31 Empire Way, including
in multiple samples under those homes. Petroleum compounds were found in the indoor
air of certain properties on the site. In terms of the levels of concentrations, these were
below GACs, other than one exceedance for benzene (based upon an IKM sample) and one
exceedance, although slight, for naphthalene. Petroleum hydrocarbon VOCs have not been
detected in any groundwater samples from the site. A high level of hydrocarbons was
detected by Monridge in a grab sample at "property X", but it has not been made clear that
it is from subsurface contamination. The soil and soil vapour readings confirm levels that
are well below the level which would be required to explain Monridge's result for property
X. In his evidence in court, Professor Eddleston observed that on Mr Quint's predicted
concentrations of hydrocarbons in houses the maxim of benzene was 1.5mg, which is much
lower than the concentration Monridge were expecting. It therefore seems clear that the
concentrations measured by others are plainly not high enough to support the Monridge
calculation.
[101]
In the "property X" sample of indoor air, taken by Monridge, the presence of MTBE
is noted. This is a substance which is an additive of unleaded petrol, introduced from
around 1986. The presence of unleaded petrol would therefore post-date the time when
39
industrial activity is known to have been active at the site. Importantly, MTBE was not
detected in any of soil samples or soil vapour samples (of which there are over 200 across
the site), or groundwater samples. This strongly suggests that, even if MTBE is detected in
indoor air, it is not coming from the ground, because if it is then it would be expected to be
picked up in the sampling.
[102]
It is true that Mr Firth's suggestion that petrol could have been brought into the
properties by the inhabitants, and thus give rise to its presence in the air inside, seems
conjectural. It was present in the air in several properties and there is no sensible
explanation why such a number of people would bring petrol into their houses. However, it
is crucial to recognise that he said this when attempting to work out how else it might have
happened. His key points are that MTBE was not present in petrol in the UK until sometime
after the industrial sites ceased to be used and the contaminants were there, and that in any
event it was never detected in the other sampling.
[103]
Taking all of the evidence into consideration, I am not satisfied that MTBE is in the
ground or that the levels of petroleum hydrocarbon identified show any risk to human
health.
Groundwater
[104]
Groundwater containing solvents can, by migration or seepages, potentially spread
around the site. WSP installed groundwater monitoring wells, including those
named SBH03, CP1 and CP2. Groundwater flow can occur through gravel, as it provides
spaces and thus is permeable. Mr Firth explained in evidence that the gravel lenses
identified in SBH03 appeared not to extend towards the properties. WSP confirmed that
view in their Stage 3 investigation, which involved drilling the two additional wells,
40
CP1 and CP2, between SBH03 and the properties. WSP concluded that the granular horizon
was not laterally continuous and that shallow groundwater was likely to be discontinuous.
Put more simply, it would not spread.
[105]
Mr Brien expressed the opinion that the made ground detected in CP1 and CP2 was
found to contain uncompacted materials which could conceivably create pathways just as
effective (or more effective) at moving groundwater than gravel bands. Also, CP1 and CP2
were only drilled to a depth of 6 metres, rather than 10 metres which was the case
with SBH03. He felt that had CP1 and CP2 been drilled deeper they could have encountered
the groundwater seepages which, since chlorinated solvents are denser than water, could
have contained chlorinated solvents. By deeper drilling, they would likely have
encountered additional water-bearing zones. In his view, the fate and transport of the
substances identified in SBH03 has not been properly investigated.
[106]
In response, Mr Firth stated that the readings and groundwater concentrations
in SBH03, in the clay layer below 6m depth where the seepages were observed, are
significantly lower than in the overlying made ground. The data indicates that the seepages
are unlikely to be associated with a migration pathway of concern.
[107]
Mr Firth has qualifications and experience in hydrogeology, and I agree with his
view that WSP's conclusions on the discontinuity of groundwater should be accepted.
However, even if these conclusions are not accepted, this is yet another example of the
pursuers' experts contending that the investigations done by WSP were in some way
inadequate and that other areas of contaminants may well exist. The fact that this raised
concerns with these experts is, of course, understandable but there is no further evidence to
show that groundwater migration is the cause of extended or higher levels of contamination.
41
Rashes
[108]
There was evidence from a number of the pursuers about suffering a rash after direct
contact with the soil, generally when doing gardening work. Several other pursuers stated
they had suffered rashes, without referring to contact with soil. There was, however, no
evidence, from Professor Douglas or others, to show that rashes were actually caused by the
contaminants found in the site. There was also no evidence to suggest that rashes could not
be picked up when in contact with soil without contaminants of the kind known to exist
here. While the direct contact point had some force in the pursuers' evidence, it has not
been proved, on the balance of probabilities, that this results in the property not being
reasonably fit for habitation.
Cancer risks and breach of duty
[109]
Certain of the experts (including Professor Douglas and Mr Firth) made reference to
the carcogenicity of TCE, that is its risk in promoting the development of cancer. For the
pursuers it was submitted that Mr Firth referred to the US EPA Regional Screening Levels
on the increased risk of cancer from TCE, which is at a level below what Mr Firth found to
be present in the properties. It was said not to be necessary to get cancer to suffer harm; the
increased risk sufficed and a pursuer could get damages for it. The defender's position is
that there is no evidence of any of the pursuers having cancer, no more than a hypothetical
risk that they could develop cancer in the future and no basis for damages to be awarded. I
will comment later on causation in relation to the cancer issues, the present point being
whether there is a cancer risk that results in a breach of the statutory duty.
[110]
While it can take some years for cancer to develop, on the evidence none of the
pursuers has suffered cancer over the years they have lived there. One other resident has
42
been diagnosed with cancer, but the cause has not been linked to the contaminants. There is
a theoretical or minimal increased risk of cancer from the amounts of TCE identified and
that does not show the properties to be not reasonably habitable. Inferring breach of duty
from such low-levels of risk is not appropriate.
Greater risk in specific properties
[111]
It is open to argument that there could be a higher level risk in specific properties
(for example, where there were hotspots) and hence for particular pursuers. I have therefore
considered the position of each individual pursuer. There was, however, no evidence
identifying in these properties a sufficient level of risk to justify a breach of duty. The
pursuers' case focusses largely on the properties of all of the pursuers and a general
exposure to contaminants which can cause harm. If actual harm is relied upon to show
breach of duty, it is noteworthy that the symptoms reported by those who lived in these
specified properties were broadly the same as those in the others. When the other properties
are habitable, but the tenants have the same symptoms (which are of course common
symptoms for the public generally) then breach of duty is not established by actual harm
only in relation to the specified properties.
Other potential contaminants
[112]
It was agreed by the experts that there could be other contaminants present within
Plot A which were not analysed in the WSP investigations. This identifies a possibility
rather than a probability. It was not confirmed by any factual evidence that they exist, nor
was there any evidence that if they do exist they would be harmful.
43
Conclusions on the level of contamination and whether it poses a risk to health
[113]
Mr Brien described part of his remit as being to establish whether there was a
problem with the conceptual site model used by WSP. He considered that there was a
problem, but he did not prepare his own model. Monridge did not fully delineate the extent
of contaminations. Potential risks could not be ruled out. Of itself, while critical of WSP,
that does not provide evidence allowing conclusions to be drawn about the level of
contaminants and the risk they could pose.
[114]
Mr Quint's report makes a number of criticisms or challenges to Mr Firth's report, to
which Mr Firth responds in his supplementary report. Mr Quint reached the view that there
was a risk to the health or safety of the occupants, but he was unable to make any
observations on the level of risk. He accepted that there could be uncertainties in using the
Monridge indoor air data, which as "snapshots" did not give any assistance about long-term
exposure.
[115]
Mr Witherington's point, that residents at the site are more likely than not to have
been exposed to higher levels of VOCs in their gardens than they would have otherwise
been exposed to if adequate investigations and remediation had been done, does not assist
in relation to risk. He confirmed in his oral evidence that he had not been instructed to
consider risk to residents and his report does not give any opinion regarding the level of
risk. He recognised that the divergence between WSP and Monridge could be caused by a
deficiency in either of their approaches.
[116]
Mr Firth recognised that WSP sampling is not representative of soil vapour
concentrations beneath properties. It is, however, clear that Mr Firth did a detailed scientific
analysis of the data, and reached an overall view about the results found by WSP and
Monridge. While I do not see Mr Firth's evidence as rebutting all that has been said by the
44
pursuers' experts, it does nonetheless establish, on the approach he took, levels of
contamination that do not show a level of risk to human health any higher than low or
tolerable levels. His analysis is criticised by, for example, Mr Quint, but even if Mr Firth's
analysis is wrong, we simply do not have in the pursuers' expert evidence on environmental
matters sufficient material to prove the opposite. Mr Brien's and Mr Witherington's points
that previous investigations had not been sufficient and more investigations would be
required do not take us any further. It was agreed that the full extent of contamination at
any individual property is not known and that while contamination levels are likely to have
been higher in the past, they cannot say by how much.
[117]
I therefore conclude from the evidence of these experts that the pursuers have not
established, on the balance of probabilities, that the level of contaminants on the site posed a
risk to human health making the development not reasonably fit for human habitation. It
remains possible, however, that the pursuers can still prove breach of duty if the evidence as
a whole shows that the harm suffered by the pursuers was caused, or materially contributed
to, by the contaminants. This was a key issue for the toxicology experts.
Assessment of toxicology experts' evidence
[118]
Before turning to assess the evidence of the experts on toxicology, it is worth noting
three important points. Firstly, there is a lack of research, and hence no evidence, to show
what the effects of chronic exposure to low-level solvent fumes from the contaminants
identified may be and in particular whether it can cause the symptoms referred to by the
pursuers. Secondly, it is not possible to know with any certainty the concentration of, or
exposure to, any particular contaminant which was present at each property at, and since,
the commencement of the tenancy. Thirdly, there was no medical evidence led that the
45
symptoms were as a matter of fact caused by the contamination, although equally there was
also no evidence that they were actually caused by something else other than contamination,
albeit there is evidence that they could have been.
Weight to be given to the pursuers' evidence
[119]
While the pursuers rely on the expert evidence of Professor Douglas, it was
mentioned only on a limited number of occasions in the written submissions and oral
submissions. Beyond what is explained below, there is no detailed analysis in the pursuers'
submissions of his reasoning or any real or convincing attempt to draw out a basis for his
evidence being seen as superior to that of Professor Eddleston. In fact, the bulk of the
pursuers' submissions rests on the evidence of the pursuers.
[120]
It was accepted for the pursuers that Professor Douglas has acknowledged the
absence of any specific study in relation to chronic low-level exposure in a residential
setting. However, it was explained that he has drawn on the work of Dr Flaks and his own
knowledge and experience in considering the medical records and the evidence from the
pursuers. By way of example, in response to a question relating to what documents he had
before him for his report on a pursuer, he said that he would have had the medical
questionnaire, the medical records and statements. He said that he did not dismiss medical
records, but they are incomplete and can miss common symptoms.
[121]
Professor Eddleston saw the medical records as providing a more reliable source of
evidence than the witness statements, although the defender accepted the generality that not
all issues are reported by individuals to their GPs. Professor Douglas accepted that there
could be a link between symptoms and people worrying about living on potentially
contaminated land, which may explain resolution when they moved out. With the
46
exception of Lee Brannan, all of the pursuers were living at the site from around 2010 when
the issue of possible contamination became public knowledge.
[122]
It was clear from the evidence of Professor Douglas that he had focused on the
pursuers' witness statements and did not appear to attach much significance to the medical
records which Professor Eddleston relied upon. While he said he had looked at them, he did
not seem to have appropriate regard to other potential causes for the symptoms, where there
were obvious alternatives and the symptoms are extremely common. In one of his reports
he drew attention to health inequalities and adverse social circumstances causing harm, but
his reports on individual pursuers took no account of those factors as potentially causative.
The pursuers' evidence was taken as read and Professor Douglas did not critically assess
whether what was narrated in the pursuers' witness statements is accurate. In particular, he
did not make much of the fact that a number of the pursuers complained of the symptoms
prior to moving to the site. Moreover, as noted when summarising his evidence earlier
(para [39]), in the symptom questionnaire based on 99 individuals there were some values
significantly lower than what was stated in the pursuers' evidence, such as stress (1%), poor
concentration (1%) and disturbed sleep (1%).
[123]
Counsel for the pursuers argued that Professor Eddleston said, in effect, that each
pursuer was making up a narrative. I disagree. Professor Eddleston did not actively
challenge the credibility of the pursuers. His approach was much more measured. The
pursuers were, as is accepted by the defender, generally doing their best to assist the court in
their evidence. It is not suggested that they were not telling the truth, rather that they gave
an account that was to some degree unreliable when one has regard to the full
circumstances.
47
[124]
There are certain points that affect the reliability of the pursuers' evidence. A fair
number of the pursuers had some of the symptoms prior to moving in. The frequency or
significance of the ailments are not the same as is recorded in the GP records. Notes taken
by GPs are contemporaneous, while the pursuers' main witness statements and evidence
were provided a number of years later. There are difficulties in accurately recalling the
timing and severity of symptoms following a significant delay. Professor Eddleston's close
examination of each person's medical history in their GP and hospital notes confirmed that
many of the problems pre-existed their time at the site or occurred afterwards. He also
noted that a number of pursuers saw their GPs frequently whilst living at the site but never
discussed the issues which now formed the basis of the claims.
[125]
It is obviously possible that symptoms which have been reported before and perhaps
not resolved by the GP's assistance might not be mentioned again, so the point made by
Professor Eddleston about frequency is not fully supported. Reasons were given by the
pursuers for not reporting symptoms, but matters of significance should and normally
would be reported to the GP when attending. However, there were also reasons given by
the pursuers for not going to the GP, for example self-medicating, and these were to some
extent understandable. In relation to Professor Eddleston's view that the pursuers who left
their tenancy felt better because of their anxiety lessening, it was pointed out to him that
some pursuers who had been elsewhere reported feeling better before contamination of the
site became common knowledge. I am not persuaded that improvement of conditions after
leaving can be wholly attributed to a decrease in health anxiety. Nevertheless, it is a
possibility and health anxiety may be one of the reasons for the differences between the
pursuers' evidence and the medical records. The main point is that a number of differences
exist.
48
[126]
Professor Eddleston's opinion, based on his expertise, was that the symptoms would
have been expected to fade immediately after the exposure finished if exposure to solvents
had been the cause, for example because TCE leaves the body within 10 days. The account
given by the majority of the pursuers is not consistent with that expert evidence, and some
of them spoke to things resolving or at least improving over months. Some force exists in
the point that several pursuers spoke to not having the symptoms before or after living in
the property, but that is to a degree contradicted by what is said and not said in their
medical records and for at least some of them it could possibly have been affected by the
anxiety consequences of having been told about the contamination.
[127]
In my opinion, while the evidence given by the pursuers was credible, there are, for
the reasons just explained, certain pointers towards a degree of unreliability, which results
in their evidence not fully being accepted. To that extent, the heavy reliance on that
evidence by Professor Douglas and counsel for the pursuers is not fully supported.
References by the pursuers to visitors or family members having symptoms when at the
property is noted, but these other people did not give evidence and the details of their
medical histories are not known. Some pursuers refer, in their witness statements, to mould
or dampness in their properties. This was not relied upon and in any event there was no
real evidence about it being linked to contaminants.
Other issues about the pursuers' evidence
[128]
Aside from the pursuers' reliability, other difficulties in their case can be identified.
Professor Eddleston explained that, had the symptoms been caused by solvent exposure
when the levels were sufficient to cause symptoms, the pursuers would have been expected
to present with multiple related symptoms at the same time, whereas the medical records
49
show different issues being presented at different times. There was no clear evidence that
the level of contaminants were the same and remained the same in respect of each property,
but I see force in this point because the thrust of the pursuers' case is continuing solvent
exposure on the site. He also said that the description given by pursuers of their symptoms
is often not consistent with solvent exposure having been the cause. For example, a number
describe headaches focussed to a particular area, which is more likely to be explained by a
different cause because a solvent induced headache is more likely to be general in nature.
This is another example of there being other potential causes of the symptoms. In addition,
he referenced the wide range of symptoms mentioned by the pursuers.
[129]
He also commented on organ damage that is known to be caused by exposure to
certain levels of solvents (cancer, liver, kidney or nerve damage) not being reported by any
of the pursuers. In other words, things which are, on the medical literature, capable of being
brought about by exposure to solvents were not mentioned here. He explained that
dementia is recognised as a consequence of exposure and some of the claimants report
cognitive or memory problems. However, these are common consequences of anxiety and
seemed to have settled after people left the development, which would not have occurred if
due to solvent-induced brain cell damage. Brain damage can be identified and measured
through neurocognitive testing, but this has not been done for any of the pursuers.
[130]
Professor Eddleston referred to workplace exposure limits being set to avoid any ill-
health in workers and so having a large safety margin. He accepted that these are not
directly applicable to the residential setting but took the view that they did provide a
relevant context for the pursuers' claims. Of course, the pursuers were not subject to the
same timings for workplace exposure, such as eight hours a day, when some spent more
time in their houses. Nonetheless, in the absence of any literature about residential
50
exposure, the workplace exposure limits, and their large safety margin, provide some
assistance. They deal with long-term exposure and take a conservative approach, as
explained by Professor Eddleston.
[131]
Much of these aspects of Professor Eddleston's reasoning have force and are not able
to be discounted by the evidence of Professor Douglas or the points made in the pursuers'
submissions.
Common symptoms, synergy and exacerbation
[132]
It is a major feature of the basis for Professor Douglas`s conclusion on causation that,
in his view, the thirteen pursuers presented with a pattern of common symptoms. However,
the evidence of Professor Eddleston undermines this approach to some degree, including
the different timings of symptoms. There is also the absence of a control group against
which to compare the common symptoms of pursuers, Professor Douglas having accepted
in response to a question from Mr Johnston KC that a control group would be desirable if
seeking to draw conclusions based upon the number of people with such symptoms. In
reaching views on the commonality of symptoms he had regard only to the thirteen
pursuers when we have no real information about the other residents on the site. There is
no statistical analysis showing that the thirteen pursuers would otherwise have been
unlikely to suffer from these symptoms. Professor Douglas places great strength on the
commonality and he was not, in my opinion, taking a sufficiently objective or rational
approach in that regard.
[133]
In relation to the potential for synergy, additionality or compound effects due to the
risk of there being more than one source of contamination exposure, Professor Douglas did
not provide any real justification for his view that the contaminants known to be present in
51
the site would have a synergistic effect in relation to the impact on health. His reference to
the combined effect of asbestos fibres and smoking causing cancer is merely an example of
an established form of synergy. The fact that there are such established examples does not
assist when no evidence was presented which would support the conclusion that the types
of contaminants and substances in the present case would have any synergistic effect.
[134]
The pursuers contend that Professor Eddleston did not give weight to the evidence
that symptoms a pursuer had experienced before could have been exacerbated by the
contaminants when in the tenancy. However, that again requires evidence of some proper
medical basis for exacerbation by contaminants of symptoms which can arise from other
causes, and there was none.
Cancer risks and causation
[135]
There is no evidence that any of the pursuers in this case has suffered cancer, let
alone whether it was caused by the contaminants. Evidence was led that another resident
had been diagnosed with cancer, but again it was not linked to the contaminants, with the
nature and cause of it not being the subject of any detailed evidence. Professor Eddleston
explained that there is no medical literature showing that a low-level exposure to TCE or
mixed solvents could cause cancer problems. While in theory it could, there is no data and
no publications. The screening levels relied upon by the pursuers are set on the basis that
the increase in risk is 1 in 100,000. Professor Eddleston stated that this is a lower screening
level than that adopted in the UK.
[136]
If there was a breach of the statutory duty, I am unable to conclude that there is a
liability in damages for a limited increase in risk of this kind. The pursuers have not shown
any proper legal basis for reaching that conclusion. Senior counsel for the defender gave the
52
valid example that while pleural plaques can give rise to damages, mere exposure to the risk
caused by asbestos does not. Accordingly, on cancer risk the pursuers do not meet the
requirements of a basis for damages.
[137]
Counsel for the pursuers also accepts that the indication in 2011 or so that cancer
could be caused is likely to have contributed to mental health and anxiety. This was
accepted and indeed relied upon by Professor Eddleston.
Conclusions on assessment of toxicology experts
[138]
Professor Eddleston is a clinical toxicologist and Professor Douglas accepted in
cross-examination that he is not, at least strictly speaking, a toxicologist. Professor Douglas
retired from his final research job in 1992 and his work since then has mainly been medical
legal work. He stated that he had reviewed Dr Flaks' report but had not read through the
literature cited therein and did not rely upon it in any detail in his own evidence, although
he noted that he knew or had met some of the authors. The approach taken by
Professor Douglas was more idiosyncratic, in that his report was much shorter than that of
Dr Flaks, not founded heavily on the literature, and he commonly made anecdotal
references to colleagues or historical medical professionals and quoted things they have
said.
[139]
Professor Douglas's evidence, while well-put, had certain shortcomings when
compared to the more forensic and robust analysis undertaken by Professor Eddleston. He
did not give full consideration to the actual levels or concentrations of contaminants found
on the site. He also did not give any real weight to the absence of symptoms of the pursuers
which would be expected from solvent exposure risks, such as cancers or damage to major
53
organs. He paid limited attention to the differences between the medical records and the
witness statements.
[140]
As said by Professor Eddleston in his oral evidence, without a control group it is
very difficult to tease out the symptoms reported by the pursuers, which are common
conditions, from what the general public have. On his view, on the balance of probabilities,
these were common illnesses, going to happen anyway, and he considers that anxiety about
being exposed to chemicals and cancer is a large part of what was reported. Even if less
weight is given to the latter point, he explains the absence of any research which establishes
a link between the symptoms and chronic low-level exposure. The pursuers accept that
there is a lack of such research. Professor Eddleston makes the strong point that as a result
there is no evidence that such exposure to solvents could cause the symptoms mentioned by
the pursuers. He also explained that there was no proof of the exposure being the actual
cause. He takes into account the agreed position that there are pathways that could link
contamination to the pursuers.
[141]
I accept that Professor Eddleston's reasoning does not completely discount certain
conflicting points and I have had regard to that when assessing his evidence as a whole. But
in a case of this complexity it cannot reasonably be expected that every contention raised in
the evidence can fully be countered. In essence, a few points differing from his reasoning
can be seen as correct, but the question is whether they undermine his general approach and
conclusions. In my view, they do not. If Professor Eddleston is wrong about the pursuers'
evidence to some degree, that does not militate the overall effect of his reasoning.
Professor Eddleston's reliance on the GP records is no doubt a feature of his approach but I
conclude that the strength of the other features in his evidence outweighs any concerns
about possible erroneous points.
54
[142]
More generally, looking at the evidence of Professor Douglas as a whole, he does not
provide any medically reasoned justification which would allow the court to make findings
supporting his conclusions. He relies heavily on the evidence of the pursuers and seeks to
draw inferences from it. He does not explain in detail how he was able to reach his view on
causation notwithstanding his acceptance that there is an absence of any published evidence
of symptoms caused by low-level chronic exposure in a residential setting. Separately,
while Professor Douglas saw a link between direct contact of soil and rashes, he did not
offer any medical reasons as to why rashes could be caused by personal contact with the
specific contaminants referred to on the site. What carries weight in expert evidence is the
reasoning and not the conclusion.
[143]
I do not regard the opinions expressed by Professor Douglas as an unsubstantiated
ipse dixit account (as was submitted for the defender) and indeed I was to some extent
impressed by his knowledge, presentation and articulation. Nevertheless, his reasoning did
not contain sufficient objective, scientific or medical grounds supporting the conclusions he
reached. When one considers the general nature and substance of Professor Douglas's
reports and the fact that Professor Douglas's conclusions are not underpinned by
appropriate reliance on relevant medical or scientific literature, I conclude that
Professor Eddleston's evidence is of greater force. I accept the bulk of Professor Eddleston`s
evidence. In any event, the evidence led for the pursuers does not provide sufficient
support. The pursuers' case cannot pass the threshold of the balance of probabilities.
Conclusions
[144]
While each of the pursuers were doing their best to tell the truth, factors were
identified by Professor Eddleston which had some impact on the reliability of their evidence.
55
The central theme of the environmental expert evidence for the pursuers was potential risks
and the need for further investigations about those risks. For several reasons, the toxicology
evidence did not provide sufficient support, including that the ailments and symptoms are
common among the public and no comparison was made, with for example a control group.
There is also an absence of research on the effects of chronic low-level exposure to
contaminants in a residential setting. The absence of that research does not exclude the
possibility of an inference that the exposure is likely to cause harm or indeed did so, but
there needs to be appropriate evidence allowing any such inference to be drawn. Seeking to
draw the inferences relied upon on behalf of the pursuers, based on their own evidence, the
environmental experts and the toxicology experts, goes too far.
[145]
The lack of sufficiently supportive evidence that the levels of contamination are
capable of causing, and are likely to have caused, the ailments and symptoms suffered by
the pursuers results in the pursuers' case failing on breach of duty and on causation.
Disposal
[146]
In each of the actions, I shall sustain the defender's pleas-in-law (numbered
differently in the various cases) in the following terms: (i) the pursuer's averments, so far as
material are unfounded in fact and the defender should be assoilzied; and (ii) the pursuer
not having suffered any loss, injury or damage through breach of statutory duty on the part
of the defender, decree of absolvitor should be pronounced.
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